摘要
通过不同负荷大鼠运动模型的建立,用透射电子显微镜观察了一般游泳训练大鼠及过度负荷大鼠心肌超微结构变化。结果表明:一般训练组心肌呈与有氧代谢相适应的生理性肥大,主要是心系数增大、线粒体增多;过度负荷组则呈现病理性改变,心肌肌丝断裂,线粒体结构模型、膜破坏,并见扩张的"二联体"结构。结果提示:适宜负荷提高心脏机能,过度负荷使心脏重塑朝病态方向转化。
After normal swimming or overload training,the rats myocardium was examined.The heart coefficients and ultrastructures indicated that myocardic changes of normal swimming group adapted to aerobic metabolize mainly in mitochondrion,while those of overload group took pathological changes in 'diad body'.This study showed that suitable workload enhanced cardiac function,while overload could only cause pathological remodeling.
出处
《武汉体育学院学报》
CSSCI
北大核心
2003年第2期47-49,共3页
Journal of Wuhan Sports University
关键词
运动负荷
心肌肥大
电子显微镜
大鼠
实验研究
运动生物化学
workload
myocardic hypertrophy
electric microscope
rat
experiment and study
exercise biochemistry
