1Lloyd-Jones D, Adams RJ,Brown TM,et al. Heart disease and stroke statistics-2010 update:a report from the ameri- can heart association[J]. Circulation, 2010, 121 (2) : 212- 215. 被引量：1
2Mun CH,Lee WT,Park KA,et al. Regulation of endothelial nitric oxide synthase by agmatine after transient global cerebral ischemia in rat brain[J]. Anat Cell Biol,2010, 43 (3) : 230-240. 被引量：1
4Hua Q,Zhu X,Li P,et al. Refined Qing Kai Ling,traditional Chinese medicinal preparation, reduces ischemic strokeinduced infarct size and neurological deficitsand increases expression of endothelial nitric oxide synthase[J]. Biol Pharm Bull, 2008,31 (4) : 633-637. 被引量：1
5Mehta SL, Manhas N, Raghubir R. Molecular targets in cerebral ischemia for developing novel therapeutics[J]. Brain Res Rev,2007,54(1) :34-66. 被引量：1
6Rao S,Lin Z, Drobyshevsky A, et al. Involvement of neuronal nitric oxide synthase in ongoing fetal brain injury following near-term rabbit hypoxia-ischemia[J]. Dev Neurosci, 2011,33 (3-4) :288-298. 被引量：1
7Kikuchi K, Kawahara K, Tanchareon S, et al. The free radical scar enger, 14 edaravone rescues rats from cerebral infarction by attenuating the release of MDA, high mobility group boxl in neuron alcells[J]. Pharmacol Exp Ther,2009,329(3) :865-874. 被引量：1
8Wang B,Cao W, Biswal S, et al. Carbon monoxide-activated Nrf2 pathway leads to protection against permanent focal cerebral ischemia[J]. Med Hypotheses, 2010,75 (2): 235-237. 被引量：1
9Imuta N, Hori O, Kitao Y, et al. Hypoxia-mediated induction of heine oxygenase type I and carbon monoxide release from astrocytes protects nearby cerebral neurons from hypoxia-mediated apoptosis[J]. Antioxid Redox Signal,2007,9(5) :543-552. 被引量：1
2[1]Goodwin LR,Francom D,Dieken FP,et al.Determination of sulfide in brain tissue by gas dialysis/ion chromatography:postmortem studies and two case reports[J].J Anal Toxicol,1989,13:105-109. 被引量：1
3[2]Savage JC,Gould DH.Determination of sulfide in brain tissue and rumen fluid by ion-interaction reversed phase high-performance liquid chromatography[J].J Chromatogr,1990,526:540-545. 被引量：1
4[3]Abe K,Kimura H.The possible role of hydrogen sulfide as an endogenous neuromodulator[J].J Neurosci,1996,16:1066-1071. 被引量：1
5[4]Wang R.Two′s company,three′s a crowd:can H2S be the third endogenous gaseous transmitter?[J].FASEB J,2002,16:1792-1798. 被引量：1
6[5]Fu R,Zhao ZQ,Zhao HY,et al.Expression of heme oxygenase-1 protein and messenger RNA in permanent cerebral ischemia in rats[J].Neurol Res,2006,28:38-45. 被引量：1
7[6]Stagliano NE,Dietrich WD,Prado R,et al.The role of nitric oxide in the pathophysiology of thromboembolic stroke in the rat[J].Brain Res,1997,759:32-40. 被引量：1
8[7]Pulsinelli WA,Brierley JB.A new model of bilateral hemispheric ischemia in the unanesthetized rat[J].Stroke,1979,10:267-272. 被引量：1
9[8]Qu K,Chen CP,Halliwell B,et al.Hydrogen sulfide is a mediator of cerebral ischemic damage[J].Stroke,2006,37:889-893. 被引量：1
10[9]Eto K,Ogasawara M,Umemura K,et al.Hydrogen sulfide is produced in response to neuronal excitation[J].J Neurosci,2002,22:3386-3391. 被引量：1
