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氧化应激在口腔癌发生中的作用 被引量:4

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摘要 氧化应激能导致细胞内DNA碱基改变、链断裂、抑癌基因失活、原癌基因的表达增高,因此其与包括口腔癌在内的许多癌变的发生有关。本文主要从口腔癌患者机体氧化还原状态的改变;吸烟、饮酒、嚼槟榔与口腔癌及氧化应激的关系;维生素、微量元素与氧化应激的关系;放化疗对口腔癌患者氧化还原状态的影响等方面对氧化应激在口腔癌发生中所起的作用作一综述。
出处 《北京口腔医学》 CAS 2009年第3期178-180,共3页 Beijing Journal of Stomatology
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  • 1Loughlin KR, Manson K, Cragnale D, et al. The use of hydrogen peroxide to enhance the efficacy of doxorubicin hydrochloride in a murine bladder tumor cell line. J Uro1,2001,165 (4) : 1300-1304. 被引量:1
  • 2Ma N, Tagawa T, Hiraku Y, et al. 8-Nitroguanine formation in oral leukoplakia, a premalignant lesion. Nitric Oxide,2006,14:137-143. 被引量:1
  • 3Beevi SS, Rasheed AM, Geetha A. Evaluation of oxidative stress and nitric oxide levels in patients with oral cavity cancer. Jpn J Clin Oncol, 2004,34(7) :379-385. 被引量:1
  • 4Loft S, Poulsen HE. Cancer risk and oxidative DNA damage in man. J Mol Med, 1996,74:297-312. 被引量:1
  • 5Petros JA, Baumann AK, Ruiz-Pesini E, et al. mtDNA mutations increase tumofi-genicity in prostate cancer. Proc Natl Acad Sci USA, 2005,102 (3) :719-724. 被引量:1
  • 6Ridnour LA, Thomas DD, Donzelli S,et al. The biphasic nature of nitric oxide resp-onses in tumor biology. Antioxid Redox Signal, 2006, 8 : 1329-1337. 被引量:1
  • 7Goetz ME, Luch A. Reactive species : A cell damaging rout assisting to, chemical carcinogens. Cancer Lett, 2008,266 ( 1 ) :73-83. 被引量:1
  • 8Kim Y J, Back SH, Bogner PN, et al. Targeting the Nrt2-Prx I pathway with selen-ium to chance the efficacy and selectivity of cancer therapy. J Cancer Mol, 2007, 3(2) :37-43. 被引量:1
  • 9Hansen JM, Moriarty-Craige S, Jones DP. Nuclear and cytoplasmic peroxiredoxin-1 differentially regulate NF-KB activities. Free Radic Biol Med ,2007,43:282-288. 被引量:1
  • 10Manoharan S, Kolanjiappan K, Suresh K, et al. Lipid peroxidation and antioxidants status in patients with oral squamous cell carcinoma. Indian J Med Res,2005, 122(6) :529-534. 被引量:1

同被引文献44

  • 1Beevi SS, Rasheed AM, Geetha A. Evaluation of oxidative stress and nitric oxide levels in patients with oral cavity cancer. Jpn J Clin Onco1,2004,34 ( 7 ) :379-385. 被引量:1
  • 2Kolanjiappan K, Ramachandran CR, Manoharan S. Biochemical changes in tumor tissues of oral cancer patients. Clin Biochem,2003, 36( 1 ) :61-65. 被引量:1
  • 3Kaspar JW, Niture SK, Jaiswal AK. Nrf2 : INrf2 ( Keapl ) signaling in oxidative stress. Free Radic Biol Med, 2009,g7(9) :1304-1309. 被引量:1
  • 4Kuo CC, Liu TW, Chen LT, et al. Combination of arsenic trioxide and BCNU synergistically triggers redox-mediated autophagic cell death in human solid tumors. Free Radio Biol Med,2011,51 (12): 2195 -2209. 被引量:1
  • 5Chien CW,Yao JH, Chang SY, et al. Enhanced suppression of tumor growth by concomitant treatment of human lung cancer cells with suberoylanilide hydroxamic acid and arsenic trioxide. Toxicol Appl Pharmacol, 2011, 257(1 ):59-66. 被引量:1
  • 6Kumar P, Gao Q, Ning Y, et al. Arsenic trioxide enhances the therapeutic efiqcacy of radiation treatment of oral squamous carcinoma while protecting bone. Mol Cancer Ther, 2008,7 (7) :2060-2069. 被引量:1
  • 7Le A, Cooper CR, Gouw AM, et al. Inhibition of lactate dehydrogenase A induces oxidative stress and inhibits tumor progression. Proc Natl Acad Sci USA, 2010,107 ( 5 ) :2037-2042. 被引量:1
  • 8Jaiswal AK. Nrf2 signaling in coordinated activation of antioxidant gene expression. Free Radic Biol Med,2004,36(10) :1199-1207. 被引量:1
  • 9Chen XL,Kunsch C. Indction of cytoprotective genes through Nri2/ antioxidant response element pathway: a new therapeutic approach for the treatment of inflammatory diseases. Curt Pharm Des, 2004, 10 (8) :879-891. 被引量:1
  • 10Hybertson BM, Gao B, Bose SK, et al. Oxidative stress in health and disease: the therapeutic potential of Nrt2 activation. Mol Aspects Med ,2011,32(4-6) :234-246. 被引量:1

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