摘要
研究急性胆道高压对犬血压、左室压力与内脏大神经冲动的关系，探讨胆心反射的传入途径。方法：利用自制胆道高压模型诱导神经冲动频率及左心室插管技术。结果：急性胆道加压并维持压力恒定（250mmHg）表现为血压及左室压下降（P＜0．01）而右内脏大神经冲动频率增加（P＜0．01）。当切断右侧及切断双侧内脏大神经后加压，血压均无明显变化。结论：胆道高压引起的血压下降及左室压降低主要传导神经是右侧内脏大神经即交感系统而非迷走神经传人。该途径可能是胆心反射的传人途径而与传统的迷走神经传入机制不同。
Objective: To study the effect of acute Biliary hypertension on blood presure,ventrical presure,and splanchnic nervous imoulse frequency,and to probe into the mechanism and pathway of biliaryheart reflex Methods to lead nervous impulse freqency and use cardiac catherization on acute biliary hypertension model of dogs. Results Pressureing the biliary tract and maintaning the pressure at 250mmHg level,then the blood pressure and left ventricular spike pressure decreased compared with original values (P<0. 01),but the right splanchnic nervous freqency increased significantly (Pr0. 01 ). While the right or double splanchnic nervou were been cutting off,then increasing biliary tract pressure the blood pressure,left ventricular pressure keep constant. Vonclusivns: the ma jor nervous pathway of high biliary tract pressure affecting blood pressure and left ventricular pressure is right splanchnic route not vagus route.
出处
《肝胆外科杂志》
1997年第3期185-187,共3页
Journal of Hepatobiliary Surgery
关键词
胆道高压
植物神经
Biliary hypertension Vegefative revous Splanchnic nervous
