摘要
结果提示:急性运动过程中线粒体离子代谢紊乱抑制线粒体氧化磷酸化过程,减少 ATP 生成,进而造成线粒体肿胀、嵴破坏,进一步抑制氧化磷酸化,加剧离子代谢紊乱的“恶性循环”可能是运动性骨骼肌疲劳的重要原因。耐力训练通过提高线粒体的代谢机能,改善运动中线粒体离子代谢,以维持其正常形态可能是耐力训练增强机体抗疲劳能力,提高运动机能的重要机制。
This paper studied not only the changes of mitochondrial calcium,magnesium metabolism and mitochondrial stereome-trydata of rats after they ran for 200 minutes,but also the effect of endurance training on the indexes mentioned above.The results showed that 24 hours after strenuous exercise,the content of mitochondrial calcium increased,magnesium con-tent decreased,the volume density,mean diameter,mean area,mean volume of mitochondria increased,and the specificsurface of mitochondria decreased for rats without previous training,which indicated the swelling of mitochondria and thesmoothness of its surface were caused by the decrease of metabolic function of mitochondria after strenuous exercise,and theinteraction of the form of mitochondria and metabolic function resulted in the fatigue of skeletal muscle.The content of mito-chondrial calcium increased,the decrease of magnesium content slowed down,and the normal form of mitochondria wasmaintained after strenuous exercise for 200 minutes for rats with eight weeks' endurance training.This showed that en-durance training might be the major mechanism for delaying fatigue and improving exercise ability.
出处
《体育科学》
CSSCI
北大核心
1993年第4期68-72,共5页
China Sport Science
关键词
运动
骨骼肌
疲劳
线粒体
exercise,endurance training,mitochondria,calcium,magnesium,stereometry
