期刊文献+

卵巢早衰中西医病因病机及治疗研究进展 被引量:9

Research progress in the etiology,pathogenesis and treatment of premature ovarian failure in traditional Chinese and western medicine
下载PDF
导出
摘要 卵巢早衰(premature ovarian failure,POF)属于妇科内分泌疾病,临床治疗较为困难。POF的发病机制复杂,中医认为与肾虚、冲任虚衰等有关,治疗当以补肾填精、调理冲任为主,临床也需辩证施治。西医则认为与遗传、免疫、环境、医源性因素等有关,治疗方法以激素替代疗法、促排卵疗法、干细胞移植等为主,临床中POF的治疗以中西医结合为常见方案。现从中西医两个角度出发,对各自的发病机制给予分析,并查阅文献汇总中西医临床治疗进展进行综述性分析。 Premature ovarian failure(POF)is a gynecological endocrine disease,which is difficult to treat clinically.The pathogenesis of POF is complex.Traditional Chinese medicine believes that it is related to kidney deficiency and deficiency exhaustion of thoroughfare and conception channels.The treatment of POF should mainly focus on invigorating kidney for consolidating semen and coordinating Chong and Conception Vessels.Syndrome differentiation of POF is also necessary in clinical practice.Western medicine believes that it is related to heredity,immunity,environment,iatrogenic factors and so on.The treatment methods are mainly hormone replacement therapy,ovulation promotion therapy,stem cell transplantation and so on.The common treatment of POF in clinical practice is the combination of traditional Chinese and western medicine.This article analyzes the pathogenesis of this disease from the perspectives of traditional Chinese medicine and western medicine,and reviews the clinical treatment progress of this disease through literature review.
作者 柳晓亮 刘一鸣 苏原 连方 LIU Xiaoliang;LIU Yiming;SU Yuan;LIAN Fang(The First Clinical Medical College,Shandong University of Traditional Chinese Medicine,Jinan 250014,China;The Affiliated Hospital of Shandong University of Traditional Chinese Medicine,Jinan 250011,China)
出处 《长春中医药大学学报》 2023年第7期811-816,共6页 Journal of Changchun University of Chinese Medicine
基金 国家自然科学基金委员会资助项目(81974577)。
关键词 卵巢早衰 中西医 临床诊治 发病机制 病因 研究进展 premature ovarian failure traditional Chinese and western medicine clinical diagnosis and treatment pathogenesis etiology research progress
  • 相关文献

参考文献30

二级参考文献293

共引文献494

同被引文献130

引证文献9

二级引证文献1

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部