1Eadie M J, Sutherland JM, Doherty RE Eneephaitis in etiology of parkin- sonism in australia[J]. Arch Neurol, 1965 ; 12 i240-5. 被引量:1
2Marttila RJ, Eskola J, Ralvarinta M, et al. Immune fane tions in Parkin- son's disease[J]. Adv Neural,1984 ;40:315-23. 被引量:1
3Marttila R J, Eskola J, Soppi E, et al. Immune functions in parkinson's disease lymphocyte subsets, concanvalin A-induced suppressor cell activi- ty and in vitro immunoglobulin production [J]. Neural science, 1985;69 (3) :121-31. 被引量:1
6Fiszer U, Mix E, Fredrikson S, et aL Gama delta cells are increased in pa- tients with Parkinson's disease[J]. J Neurol Sci, 1994;121 (1) :39-45. 被引量:1
7Boka G, Anglade P, Wallach D, et al. Immunocytoehemical analysis of tumor necrosis factor and its reeepors in Parkinson's disease[J]. Neu- rosci Lett,1994;172(1-2) :151-4. 被引量:1
8Le WD, Rowe DB, Jankovic J, et al. Effects of eerebrospinal fluid from patients with Parkinson's disease on dopaminergic ceils[J]. Arch Neu- ro1,1999;56(2) :194-200. 被引量:1
9Kawaguchi Y, Kovacs J J, McLaurin A, et al. The deacetylase HDAC6 reg- ulates aggresome formation and cell viability in response to misfolded pro- tein stress[J]. Cell.2003:115(6) .727-38. 被引量:1
10Nishmura M. Tumor necrosis factor gene polymorphisms in patients with sporadic Parkinson's disease[J]. Neurosci Lett,2001 ;311 (1) :1-4. 被引量:1
二级参考文献51
1[2]Kruger R, Kuhn W, MUller T, et al. Ala 30 promutation in the gene encoding α-synuclein in Parkinson's disease. Nature Genet,1998, 18(2): 106-108. 被引量:1
2[3]Volles MJ, Less SJ, Rochet JC,et al. Vesicle permeabilization by protofibrillar alpha-synuclein, implications for the pathogenesis and treatment of Parkinson's disease. Biochemistry,2001, 40(26): 7812-7819. 被引量:1
3[4]Junn E, Mouradian MM. Human alpha-synuclein over-expression increase intracellular reactive oxygen species levels and susceptibilility to dopamine. Neurosci Lett, 2002, 320(3): 146-150. 被引量:1
4[5]Lee FJ, Liu F,Pristupa ZB, et al. Direct binding and functional coding of α-synuclein to the dopamine transporters accelerate dopamine-induced apoptosis. FASEB J, 2001, 15(6): 916-926. 被引量:1
5[6]Ball J. Current advances in Parkinson's disease. Trends Neurosci,2001, 24(7): 367. 被引量:1
6[8]Liss B, Roeper J. Molecular physiology of neuronal K +-ATP channels. Mol Membr Biol, 2001, 18(2): 117. 被引量:1
7[9]Bharath S, Hsu M, Kaur D, et al. Glutathione,iron and Parkinson's disease. Biochem Pharmacol, 2002, 64(5-6): 1037. 被引量:1
8[12]Viswanath V, Wu Y, Boonplueang R, et al. Caspase-9 activation results in downstream Caspase-8 activation and bid cleavage in 1-methyl-4-phenyl-1, 2, 3, 6-tetrahydropyridine-induced Parkinson's disease. J Neurosci, 2001, 21 (24): 9519. 被引量:1
9[13]Le WD, Rowe DB. Effects of cerebrospinal fluid from patients with Parkinson's disease on dopaminergic cells. Arch Neurol,1999, 56(2): 194. 被引量:1
10[14]Nagatsu T, Mogi M, Ichinose H, et al. Changes in cytokines and neurotrophins in Parkinsons' disease. J Neurol Transm Suppl,2000,60: 277-290. 被引量:1