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线粒体在肺纤维化发病机制中的作用及进展 被引量:2

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出处 《中国实用医药》 2010年第2期241-243,共3页 China Practical Medicine
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参考文献14

  • 1Shinfizu S, Tsujimoto Y. Proapoptotic BH3-only Bcl-2 family membersinduce cytochrome crelease, but not mitochondrial membrane potential loss. and do not directly modulate voltage dependent anion channel activity. Proc Natl Acad Sci USA,2000,97 ( 2 ) : 577-582. 被引量:1
  • 2刘朝巍,张涛,杨卓.氧化应激损伤线粒体参与癫痫病理过程[J].中国病理生理杂志,2008,24(1):198-200. 被引量:20
  • 3Lin HH, Hsu HL, Yeh NH. Apoptotic cleavage of NuMA at the Cterminal end is related to nuclear disruption and deathamplification.Biomed Sci,2007, (14) :681-694. 被引量:1
  • 4Crow MT, Mani K, Nam Y J, et al. The mitochondrial death pathway and cardiac myocyte apoptosis. Circ Res,2004,95 (10) :957-970. 被引量:1
  • 5White SR, Dorscheid DR. Corticosteroid2induced apoptosisof airway epithelium : a potential mechanism for chronic airway epithelial damage in asthma. Chest,2002,122 (6) :278-284. 被引量:1
  • 6Biosvieux-Ulrich E, Sourdeval M, Marano F. CD437, a synthetic retinoid, induces apoptosis in human respiratory epithelial cells viacaspase-independent mitochondrial and caspase-8-dependant pathways both upregulated by JN Ksignaling pathway. Exp Cell Res, 2005.307 ( 1 ) :76-90. 被引量:1
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  • 8Wei MC, Zong WX, Cheng EH, et al. Proappoptotic BAX and BA K: a requisite gatewayto mitochondrial dysfunctionand death. Science,2001,292 (5517) :727-730. 被引量:1
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二级参考文献35

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二级引证文献11

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