摘要
刘承宜等人提出了低强度激光疗法的生物信息模型（ＢＩＭＬ）。根据ＢＩＭＬ，绿、兰和紫等冷色兴奋Ｇｓ蛋白介导的生理过程；红、橙和黄等暖色兴奋Ｇｑ或Ｇｉ蛋白或受体关联蛋白酶介导的生理过程。本文将激光的生物效应分为直接效应和间接效应两类，根据ＢＩＭＬ成功地解释了已知第二信使生理功能的系统的低强度激光照射的作用机理和临床应用，可望用于开发低强度激光疗法的临床新应用。
Liu CY et al have put forward the biological information model on low intensity laser irradiation (BIML). According to BIML, low intensity laser irradiation couples with intracellular messenger through the chromophore absorption in the cell membrane: hotcolor(red, orange, or yellow) laser irradiation activates cAMP phosphodiesterase through Gi protein, or activates phosphoinositide phospholipase C(PLC) through Gq protein, or activates one of receptor-associated kinases:cAMP↓; coldcolor(green, blue, or violet) laser irradiation activates adenylate cyclase through Gs protein: cAMP↑. In this paper, laser effects were classified into two kinds: direct effects and indirect effects and BIML was Successfully applied to studying the mechanism of intravascular HeNe laser irradiation on blood and its clinic applications.
出处
《激光生物学报》
CAS
CSCD
1998年第2期81-85,共5页
Acta Laser Biology Sinica
基金
国家自然科学基金
广东省自然科学基金
关键词
低强度
激光疗法
机理
临床应用
low intensity laser therapy mechanism clinic
