摘要
植物系统获得性抗性(Systemic Acquired Resistance,SAR)是一种能够诱导植物持续抵御病原微生物侵害的一种防御机制。SAR需要信号分子水杨酸(Salicylic Acid,SA)参与,并与能够提高抗性的病程相关蛋白(PR)的积累有关。通过对模式植物拟南芥的研究发现,异分支酸合成酶途径是合成SA的主要途径。正调控因子NPR1与转录因子TGA相互作用,诱导防卫基因表达,进而激发SAR反应。最新研究表明,脂质分子有可能是SAR反应中的移动信号分子。这些研究结果有助于进一步了解SAR反应。
Systemic acquired resistance （SAR） is a defense mechanism which is able to induce plants to resist against microorganisms continuously. SAR needs the signal molecule salicylic acid （SA） to participate in, and it is associated with accumulation of pathogenesis-related proteins （PR）, which are thought to contribute to increase the resistance. By using the model plant Arabidopsis, it was discovered that the isochorismate approach is the major source of SA. The positive regulator protein NPR1 could interact with TGA transcription factors to induce defense gene expression, which could stimulate SAR response. The latest study showed that the mobile signal for SAR might be a lipid molecule. The results of these studies would help us further understand the SAR response.
出处
《东北农业大学学报》
CAS
CSCD
2008年第12期113-117,共5页
Journal of Northeast Agricultural University
基金
国家高技术"863"资助项目(2003AA207120)
