摘要
急性胆道感染后１２小时开始，肝Ｋｕｐｆｆｅｒ细胞（ＫＣ）对１４Ｃ标记的活大肠杆菌吞噬功能持续下降，而肺泡巨噬细胞（ＡＭ）吞噬功能进行性上升。ＫＣ在感染后１２小时肿瘤坏死因子（ＴＮＦ）分泌达到高峰，此后变得耐受，ＡＭ在感染１２小时后ＴＮＦ分泌持续上升，并保持在高水平直至４８小时。
welve hours since the onset of acute cholangitis the phagocytic function of
hepaticKupffer cells on the 14C-beled living B.coli diminished progressively, but that of
pulmonaryalveolar macrophages increased progressively. 12 hours after the infection, the
tumor necrosisfactor (TNF)secreted by Kupffer cells reached its peak level,and there after
becametolerated, whereas TNF secreted by pulmonary alveolar macrophages increased
continuouslyand remained at high level up to 48 hours.
出处
《中华消化杂志》
CAS
CSCD
北大核心
1995年第6期317-318,共2页
Chinese Journal of Digestion
