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The activities of endothelial elastase and cathepsin G in rats at oxidative stress caused by heavy metals salts

The activities of endothelial elastase and cathepsin G in rats at oxidative stress caused by heavy metals salts
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摘要 CoCl2 introduction increased cathepsin G activity in the heart and liver as well as endothelial elastase (EEl) in kidney that indicated the development of destructive processes. CoCl2 introduction decreased EEl and cathepsin G activities in blood serum and cathepsin G in lungs. HgCl2 injection decreased EEl in blood serum, heart, liver, kidney and cathepsin G in blood serum. These decreasing of proteinases activities may be caused by cytotoxic effects of heavy metals and/or the inclusion of these proteases in the destructive processes and absence of their synthesis and/or release. CoCl2 introduction increased cathepsin G activity in the heart and liver as well as endothelial elastase (EEl) in kidney that indicated the development of destructive processes. CoCl2 introduction decreased EEl and cathepsin G activities in blood serum and cathepsin G in lungs. HgCl2 injection decreased EEl in blood serum, heart, liver, kidney and cathepsin G in blood serum. These decreasing of proteinases activities may be caused by cytotoxic effects of heavy metals and/or the inclusion of these proteases in the destructive processes and absence of their synthesis and/or release.
出处 《Advances in Biological Chemistry》 2013年第2期208-214,共7页 生物化学进展(英文)
关键词 ENDOTHELIAL ELASTASE CATHEPSIN G Cobalt CHLORIDE Mercury CHLORIDE RATS Endothelial Elastase Cathepsin G Cobalt Chloride Mercury Chloride Rats
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