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胃MALT淋巴瘤发病机制研究进展 被引量:4

Research advances in the pathogenesis of MALT gastric lymphoma
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摘要 胃粘膜相关淋巴组织 (MALT)型结外边缘区B细胞淋巴瘤通常是在幽门螺杆菌 (Hp)感染基础上发展起来的。Hp感染可引起淋巴细胞浸润 ,使胃粘膜获得MALT。在Hp刺激、自身抗原刺激和T细胞辅助下 ,浸润的B细胞活化增殖 ,偶尔发生遗传学异常进而向恶性转化。研究发现Hp感染是胃MALT淋巴瘤发生的重要病因。现综述根治Hp的临床研究及抗原选择、自身免疫在淋巴瘤发病中的作用。
作者 叶月芳 周韧
出处 《国外医学(肿瘤学分册)》 2003年第4期305-308,共4页 Foreign Medical Sciences (Cancer Section)
基金 浙江省卫生厅专项基金 (2 0 0 2ZX0 10 ) 浙江省教委科学研究基金 (2 0 0 2 0 82 1)
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