摘要
Dear Editor, Upon recognition of pathogen effectors, plant resist- ance （R） proteins trigger strong defense responses that restrict the growth and spread of pathogens. Most R pro- teins are nucleotide-binding （NB） and leucine-rich repeat （LRR） domain-containing proteins （NLRs） sharing struc- tural similarity with animal NOD-like receptors （Dodds and Rathjen, 2010）. SNC1 （Suppressor of nprl, constitutive 1） is an Arabidopsis Toll/interleukin-1 Receptor （TIR）-type NLR that was originally identified through a gain-of-function autoimmune mutant, sncl, from a forward genetic screen.
Dear Editor, Upon recognition of pathogen effectors, plant resist- ance （R） proteins trigger strong defense responses that restrict the growth and spread of pathogens. Most R pro- teins are nucleotide-binding （NB） and leucine-rich repeat （LRR） domain-containing proteins （NLRs） sharing struc- tural similarity with animal NOD-like receptors （Dodds and Rathjen, 2010）. SNC1 （Suppressor of nprl, constitutive 1） is an Arabidopsis Toll/interleukin-1 Receptor （TIR）-type NLR that was originally identified through a gain-of-function autoimmune mutant, sncl, from a forward genetic screen.
