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温郁金二萜类化合物C对急性幽门螺杆菌感染诱导胃上皮细胞炎性反应和肠上皮化生保护作用的体外研究 被引量:1

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摘要 在肠上皮化生过程中,胃黏膜分化标志物与转录因子黏蛋白5AC、性别决定区Y框蛋白2表达水平逐渐降低,而肠黏膜分化标志物尾型同源盒转录因子2、黏蛋白2表达水平逐渐上升[1-2].H.pylori感染可致胃黏膜炎性反应持续存在[3].温郁金二萜类化合物C对NF-κB通道具有抑制作用[4].
出处 《中华消化杂志》 CAS CSCD 北大核心 2015年第1期46-47,共2页 Chinese Journal of Digestion
基金 浙江省自然科学基金(LY12H29002) 浙江省中医药科学研究基金(2011ZB032)
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