1[1]Berg S,Hovda DA,Shalmon E,et al. Cerebral hyperglyc olysis following severe traumatic brain injury in humans: a position emission tomography study. J Neurosurg 1997; 86:241 被引量：1
2[2]Goodman JC,Woo SL,Colak A,et al. Lactate and excita tory amino acids measured by microdialysis are decreased by pentobarbital coma in head-injured patients. J Neurotrauma 1996; 10:549 被引量：1
3[3]Angeleri F,Majkowski J,Caechio G,et al. Post-traumat ic epilepsy risk factors: One-year prospective study after head injury. Epilepsia 1999; 40 (9): 1222 被引量：1
5[5]Yokoi I, Mori A, Kabuto H, et al. Adenosines scavenged hydroxyl radicals and prevented posttraumatic epilepsy. Free Radio Biol Med 1995; 19 (4): 473 被引量：1
6[6]Nisson P, Riley T, Norton FR, et al. Epileptic seizure activity in the acute phase following cortical impact trauma in rat. Brain Res 1994; 637:227 被引量：1
7[7]Hoeppner TJ. Cotrol of scar formation in experimentally induced epilepsy. Exp Neurol 1986; 94 (3): 519 被引量：1
8[8]Sherwin AL. Neuroactive amino acids in focally epileptic human brain. Neurochem Res 1999; 24 (11): 1387 被引量：1
9[9]Ding R, Asada H, Obata K. Changes in extracellular glutamate and GABA levels in hippocampal CA3 areas and the induction of glutamic acid decarboxylase-67 in dentate granule cells of rats treated with kanic acid. Brain Res 1998; 800 (1): 105 被引量：1
10[10]Meldrum BS. The role of glutamate in epilepsy and oth er CNS disorders. Neurology 1994; 44 (11 Suppl 8): s14 被引量：1
