摘要
To the editor： Acute myocardial infarction （AMI） and acute myeloid leukemia （AML） are two intensive and life threateningdiseases. Recent human studies demonstrated that C reactive protein （CRP）, the best characterized biomarker of inflammation, contributed to increase cardiovascular risk and AML. Moreover, CRP could stimulate the generation of reactive oxygen species （ROS）,3 which have been detected in a variety of diverse pathologic states, including atherosclerosis, and acute myeloid malignancies.
To the editor： Acute myocardial infarction （AMI） and acute myeloid leukemia （AML） are two intensive and life threateningdiseases. Recent human studies demonstrated that C reactive protein （CRP）, the best characterized biomarker of inflammation, contributed to increase cardiovascular risk and AML. Moreover, CRP could stimulate the generation of reactive oxygen species （ROS）,3 which have been detected in a variety of diverse pathologic states, including atherosclerosis, and acute myeloid malignancies.
