摘要
目的:探讨蛛网膜下腔出血后迟发脑血管痉挛的临床特点。方法:回顾性分析我院2005年2月～2009年3月收治的蛛网膜下腔出血后迟发脑血管痉挛患者60例临床资料。结果:DCVS在SAH后3～4d发生,于第2周达高峰,至少持续40d。SAH并DCVS的主要病因是颅内动脉瘤破裂,与动脉瘤的部位及大小无明显关系,SAH后基底池内的高密度影与DCVS的发生密切相关,DCVS的发生与年龄、性别无关。结论:基底池中凝血块的存在是产生DCVS的直接原因。DCVS的发生于第2周达高峰,尼莫地平持续静脉推注能显著降低SAH的死亡率。
Objective：To explore the clinical features of delayed subarachnoid hemorrhage cerebral vasospasm.Methods：A retrospective method was used to compare 60 cases of subarachnoid hemorrhage with delayed cerebral vasospasm in our hospital from February 2005 to March 2009.Results：DCVS occurred 3-4 days after the SAH,reached a peak in the second week,lasted at least 40 days.SAH complicated with DCVS was the main cause of the intracranial aneurysm rupture,and there was no significant relationship with the location and size of the aneurysm.The high density substrate pool of the SAH was closely related with the DCVS,the occurrence of the DCVS has no relationship with the patiens＇ age and sex.Conclusion：The sludged blood of the base pool is the direct cause to produce clot DCVS.The peak of the DCVS occurres in the second week,continuous intravenous injection of nimodipine can significantly reduce the mortality rate of SAH.
出处
《中国医药导报》
CAS
2011年第6期53-54,共2页
China Medical Herald
