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小胶质细胞在放射性脑病中作用的研究进展 被引量:2

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摘要 放射性脑病(radiation encephalopathy,RE)是脑组织经电离辐射后产生的以神经系统损害为主的疾病.RE的发生与放射源、放射总剂量、分割方式、受照体积以及个体因素等有关.
出处 《中华放射医学与防护杂志》 CAS CSCD 北大核心 2010年第5期617-620,共4页 Chinese Journal of Radiological Medicine and Protection
基金 国家自然科学基金(30600164)
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  • 1Theele DP,Streit WJ.A chronicle of microglial ontogeny.Glia,1993,7(1):5-8. 被引量:1
  • 2Brown H,Kozlowski R,Perry H.The importance of ion channels for macrophage and microglial activation in vitro.Glia,1998,22(1):94-97. 被引量:1
  • 3Fordyce C,Jagasia R,Zhu X,et al.Microglia Kv1.3 channels contribute to their ability to kill neurons.J Neurosci,2005,25(31):7139. 被引量:1
  • 4Koizumi S,Shigemoto-Mogami Y,NASU-TADA K,et al.UDP acting at P2Y6 receptors is a mediator of microglial phagocytosis.Nature,2007,446(7139):1091-1095. 被引量:1
  • 5Haynes S E,Hollopeter G,Yang G,et al.The P2Y12 receptor regulates microglial activation by extracellular nucleotides.Nat Neurosci,2006,9(12):1512-1519. 被引量:1
  • 6Dheen S T,Kaur C,Ling E A.Microglial activation and its implications in the brain diseases.Curr Med Chem,2007,14(11):1189-1197. 被引量:1
  • 7Van Eldik L,Thompson W,Ranaivo H,et al.Glia proinflammatory cytokine upregulation as a therapeutic target for neurodegenerative diseases:function-based and target-based discovery approaches.Int Rev Neurobiol,2007,82:277-296. 被引量:1
  • 8Limoli C L,Giedzinski E,Baure J,et al.Altered growth and radiosensitivity in neural precursor cells subjected to oxidative stress.Int J Radiat Biol,2006,82(9):640-647. 被引量:1
  • 9Kyrkanides S,Moore AH,Olschowka JA,et al.Cyclooxygenase2 modulates brain inflammation-related gene expression in central nervous system radiation injury.Brain Res Mol Brain Res,2002,104(2):159-169. 被引量:1
  • 10Rola R,Zou Y,Huang T,et al.Lack of extracellular superoxide dismutase (EC-SOD) in the microenvironment impacts radiationinduced changes in neurogenesis.Free Rad Biol Med,2007,42(8):1133-1145. 被引量:1

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