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急性一氧化碳中毒迟发性脑病研究进展 被引量:4

Research progress on delayed encephalopathy after acute carbon monoxide poisoning
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作者 葛桂荣
出处 《中国职业医学》 CAS 北大核心 2010年第5期427-428,430,共3页 China Occupational Medicine
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  • 2金泰廙主编..职业卫生与职业医学[M].北京:人民卫生出版社,2003:630.
  • 3DAWSON T M, SNYDER S H. Gases as biological messengers:nitric oxide and carbon monoxide in the brain [ J]. J Neurosci, 1994,14 (9) : 5147 -5159. 被引量:1
  • 4ISCHIROPOULOS H,BEERS M F, OHNISHI S T, et al. Nitric oxide production and perivaseular nitration in brain after carbon monoxide poisoning in the rat[ J]. J Clin Invest, 1996,97 (10) :2260 - 2267. 被引量:1
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  • 6STEPHENSON D, YIN T, SMALSTIG E B, et al. Transcription factor nuclear factor-kappa B is activated in neurons after focal cerebral ischemia [ J]. J Cereb Blood Flow Metab,2000,20(3 ) :592 -603. 被引量:1
  • 7THOM S R, BHOPALE V M, FISHER D, et al. Delayed neuropathology after carbon monoxide poisoning is immune-mediated[ J]. Proc Natl Acad Sci USA,2004,101 (37) :13660-13665. 被引量:1
  • 8FRIDKIS-HARELI M,STEILN J N, FUGGER L, et al. Synthetic peptides that inhibit binding of the myelin basic protein 85-99 epitope to multiple sclerosis-associated HLA-DR2 molecules and MBP-specifie T- cell responses[ J ]. Hum Immunol,2001,62 (8) :753 - 776. 被引量:1
  • 9MALOY K J, SALAUN L, CAHILL R, et al. CD4^+ CD25^+ TR cells suppress innate immune pathology through cytokine-dependent mechanisms [ J ]. J Exp Med ,2003,197 ( 1 ) : 111 - 119. 被引量:1
  • 10TISCHNER D, WEISHAUPT A, WAN DEN BRANDT J, et al. Polyclonal expansion of regulatory, T ceils interferes with effector cell migration in a model of multiple sclerosis [ J ]. Brain, 2006,129 (PtlO) : 2635 - 2647. 被引量:1

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