期刊文献+

牙周炎发病过程中的细胞凋亡 被引量:3

原文传递
导出
摘要 凋亡是细胞程序性死亡,发生在发育过程中和生理状态下。细胞凋亡(apoptosis)的异常与疾病的发生、发展和预后关系密切。凋亡由细胞内凋亡信号通路,即内在通路和外在通路介导。
作者 梁敏 凌均棨
出处 《中华口腔医学杂志》 CAS CSCD 北大核心 2010年第9期573-576,共4页 Chinese Journal of Stomatology
  • 相关文献

参考文献42

  • 1Hengartner MO.The biochemistry of apoptosis.Nature,2000,407 (6805):770-776. 被引量:1
  • 2Holcik M,La Casse EC,Mackenzie AE,et al.Apoptesis in health and disease.Cambridge University Press,2005:11. 被引量:1
  • 3Puthalakath H,Strasser A.Keeping killers on a tight leash:transcriptional and post-translational control of the pro-apoptotic activity of BH3-only proteins.Cell Death Differ,2002,9 (5):505-512. 被引量:1
  • 4Lavrik IN,Golks A,Krammer PH.Caspases:pharmacological manipulation of cell death.J Clin Invest,2005,115(10):2665-2672. 被引量:1
  • 5Bouchier-Hayes L,Lartigue L,Newmeyer DD.Mitochondria:pharmacological manipulation of cell death.J Clin Invest,2005,115(10):2640-2647. 被引量:1
  • 6曹采方主编..临床牙周病学[M].北京:北京大学医学出版社,2006:416.
  • 7Leone CW,Bokhadhoor H,Kuo D,et al.Immunization enhances inflammation and tissue destruction in response to Porphyromonas gingivalis.Infect Immun,2006,74 (4):2286-2292. 被引量:1
  • 8Liu R,Bal HS,Desta T,et al.Diabetes enhances periodontal bone loss through enhanced resorption and diminished bone formation.J Dent Res,2006,85 (6):510-514. 被引量:1
  • 9Ekuni D,Tomofuji T,Yamanaka R,et al.Initial apical migration of junctional epithelium in rats following application of lipopolysaccharide and proteases.J Periodontol,2005,76(1):43-48. 被引量:1
  • 10Vitkov L,Krautgartner WD,Hannig M.Bacterial internalization in periodontitis.Oral Microbiol Immunol,2005,20 (5):317 -321. 被引量:1

二级参考文献16

  • 1Lalla E, Lamster IB, Feit M, et al. Blockade of RAGE suppresses periodontitis-associated bone loss in diabetic mice[J]. J Clin Invest, 2000, 105 (8) : 1117-1124. 被引量:1
  • 2Mabamed DA, Marleau A, Alnaeeli M, et al. G (-) anaerobesreactive CD4^+ T-cells trigger RANKL-mediated enhanced alveolar bone loss in diabetic NOD mice[J]. Diabetes, 2005, 54 (5): 1477-1486. 被引量:1
  • 3Gamonal J, Bascones A, Acevedo A, et al. Apoptosis in chronic adult periodontitis analyzed by in situ DNA breaks, electron microscopy, and immunohistochemistry[J]. J Periodontol, 2001, 72 (4) : 517-525. 被引量:1
  • 4Otton R, Soriano FG, Verlengia R, et al. Diabetes induces apoptosis in lymphocytes[J]. J Endocrinol, 2004, 182(1):145-156. 被引量:1
  • 5Sugiyama T, Kobayashi M, Kawamura H, et al. Enhancement of P2X (7)-induced pore formation and apoptosis: An early effect of diabetes on the retinal microvasculature[J]. Invest Ophthalmol Vis Sci, 2004, 45(3):1026-1032. 被引量:1
  • 6Liu R, Desta T, He H, et al. Diabetes alters the response to bacteria by enhancing fibroblast apoptosis[J]. Endocrirtology, 2004, 145 (6) : 2997-3003. 被引量:1
  • 7Graves DT, Liu R, Alikhani M, et al. Diabetes-enhanced inflammation and apoptosis-impact on periodontal pathology[J]. J Dent Res, 2006, 85(1): 15-21. 被引量:1
  • 8Liu R, Bal HS, Desta T, et al. Tumor necrosis factor-alpha mediates diabetes-enhanced apoptosis of matrix-producing cells and impairs diabetic healing[J]. Am J Pathol, 2006, 168(3):757-764. 被引量:1
  • 9Liu R, Bal HS, Desta T, et al. Diabetes enhances periodontal bone loss through enhanced resorption and diminished bone formation[J]. J Dent Res, 2006, 85(6):510-514. 被引量:1
  • 10He H, Liu R, Desta T, et al. Diabetes causes decreased osteoclastogenesis, reduced bone formation, and enhanced apoptosis of osteoblastic cells in bacteria stimulated bone loss[J]. Endocrinology, 2004, 145 (1) :447-452. 被引量:1

共引文献9

同被引文献25

  • 1刘洪臣.老年口腔医学进展[J].中华老年口腔医学杂志,2003,1(1):2-4. 被引量:253
  • 2Chai J,Du C,Wu J,et al.Structeral and biochemical basis ofapoptotic activation by SMAC/DIABLO[J].Nature,2000,406(6798):855-862. 被引量:1
  • 3Shoshan Barmatz V,Kein N,Abu-Hamad S,et al.Apoptosis isregulatedby the VDAC N-terminal region and by VDAC oligome-rization:release of cytochrome C AIF and Smac/Diablo[J].Biochim Biophys Acta,2010,1797:1281-1291. 被引量:1
  • 4Li P,Nijhawan D,Budihardjo J,et al.Cytochroma and dATP-dependent formation of Apaf-1/Caspase-9 complex initiates anapoptotic protease cascade[J].Cell,1997,91(4):479-89. 被引量:1
  • 5Pan G,O’Rourke K,Dixit M.Caspase-9 bcl-x and apaf-1form fermary complex[J].Biol Chem,1998,273(10):5841-5845. 被引量:1
  • 6Leone CW,Bokhadhoor H,Kuo D,et al.Immunization en-hance inflammation and tissue destruction in response to Porphy-romonas gingivlis Infect[J].Infect Immun,2006,74(4):2286-2292. 被引量:1
  • 7Nechushtan A,Smith CL,Lamensdorf I,et al.Bax and Bak co-alesce into novel Mi-associated clusters during apoptosis[J].Cell Biol,2001,153:1265. 被引量:1
  • 8Guo Y, Nguyen K A, Potempa J. Dichotomy of gingipains action as virulence factors: from cleaving substrates with the precision of a surgeon's knife to a meat chopper-like brutal degradation of proteins[J]. Periodontol 2000, 2010, 54(1): 15-44. 被引量:1
  • 9A1-Mashat H A, Kandru S, Liu R, et al. Diabetes enhances mRNA levels of proapoptotic genes and caspase activity, which contribute to impaired healing[J]. Diabetes, 2006, 55 (2): 487-495. 被引量:1
  • 10Xu J H, Zhao Y Y, Wang J K, et al. Effects of mouse re- combinant bone morphogenetic protein-7 transfection on cell apoptosis, NF-kappaB, and downstream genes in cultured primary cardiomyocytes after simulated isehemia and reper- fusion injury [J]. J Cardiovasc Pharmacol, 2010, 56 (1): 69-77. 被引量:1

引证文献3

二级引证文献4

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部