期刊文献+

甲状腺激素对心血管系统的作用机制及临床应用 被引量:5

下载PDF
导出
摘要 甲状腺激素对心血管系统有明显的作用。且研究发现因受到心脏手术打击的重症患者都不同程度地存在着甲状腺激素水平的改变。即所谓的甲状腺病态综合征[1]:甲状腺素(T4)水平正常或稍下降,三碘甲状腺氨酸(T3)水平明显下降,而促甲状腺激素(TSH)水平正常。...
作者 李宏
出处 《心功能杂志》 1999年第1期39-41,共3页
  • 相关文献

同被引文献21

  • 1Simpson P, Savion S. Differentiation of rat myocytes in single cell cuhures with and without proliferating nonmyocardial cells. Cross-striations, ultrastructure, and chronotropic response to isoproterenol [J]. Circ Res, 1982, 50(1):101-116. 被引量:1
  • 2Cao X, Kambe F, Moeller LC, et al. Thyroid hormone induces rapid activation of Akt/protein kinase B-mammalian target of rapamycin-p70S6K cascade through phosphatidylinositol 3-kinase in human fibroblasts[J]. Mol Endocrinol, 2005, 19(1) :102-112. 被引量:1
  • 3Matsui T, Nagoshi T, Rosenzweig A. Akt and PI 3- kinase signaling in cardiomyocyte hypertrophy and survival[J]. Cell Cycle, 2003, 2(3):220-223. 被引量:1
  • 4Kinugawa K, Yonekura K, Ribeiro RC, et al. Regulation of thyroid hormone receptor isoforms in physiological and pathological cardiac hypertrophy[J]. Circ Res, 2001, 89(7):591-598. 被引量:1
  • 5Wang B, Ouyang J, Xia Z. Effects of triiodo thyronine on angiotensin-induced cardiomyocyte hypertrophy: reversal of increased beta myosin heavy chain gene expression[J]. Can J PhysiolPharmacol, 2006, 84(8-9):935-941. 被引量:1
  • 6McMullen JR, Sherwood MC, Tarnavski O, et al. Inhibition of mTOR signaling with rapamycin regresses established cardiac hypertrophy induced by pressure overload[J]. Circulation, 2004, 109(24):3 050-3 055. 被引量:1
  • 7Shioi T, McMullen JR, Tarnavski O, et al. Rapamycin attenuates load-induced cardiac hypertrophy in mice [J]. Circulation, 2003, 107(12):1 664-1 670. 被引量:1
  • 8Richardson CJ, Schalm SS, Blenis J. PI3-kinase and TOR: PIKTORing cell growth [J]. Semin Cell Dev Biol, 2004, 15(2):147-159. 被引量:1
  • 9Kuzman JA, Vogelsang KA, Thomas TA, et al. L- Thyroxine activates Akt signaling in the heart[J]. J Mol Cell Cardiol, 2005, 39(2):251-258. 被引量:1
  • 10McMullen JR, Shioi T, Huang WY, et al. The insulin-like growth factor 1 receptor induces physiological heart growth via the phosphoinositide 3-kinase (p110alpha) pathway[J]. J BiolChem, 2004, 279(6): 4 782-4 793. 被引量:1

引证文献5

二级引证文献7

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部