4[1] Moller D E,Chang P Y, Yaspclkis B B, et al. Transgenic mice with muscle-specific insulin resistance develop increased adiposity: impaired glucose tolerance, and dyslipidemia [J]. Endocrinology, 1996,137(6):2397～2405. 被引量：1
5[2] Terauchi Y, Iwamoto K, Tamemoto H, et al. Development of non-insulin-dependent diabetes mellitus in the double knockout mice with disruption of insulin receptor substrate-1 and beta cell glucokinase genes. Genetic reconstitution of diabetes as a polygenic disease [J].J Clin Invest, 1997,99(5):861～686. 被引量：1
6[3] Rajkumar K, Dheen S T,Murphy L J. Hyperglycemia and impaired glucose tolerance in IGF binding protein-1 transgenic mice[J]. Am J Physiol，1996,270(4 p 01):E 565～571. 被引量：1
7[4] Ko G T, Chan J C, Wioo J et al. Serum bilirubin and cardiovascular risk factors in a Chinese population [J]. J Cardiovasc Risk, 1996,3(5):459～463. 被引量：1
8[5] Vijayalingam S, Parthiban A, Shanmuasundaram K R, et al. Abnormal antioxidant status in impaired glucose tolerance and noninsulin-dependent diabetes mellitus [J]. Diabet Med, 1996,13(8):715～719. 被引量：1
9[6] Haffner S M, Agil A, Mykkanen L, Stern M P, et al. Plasma oxidizability in subjects with normal glucose tolerance, jmpairedglucose tolerance, and NIDDM [J]. Diaetes Care, 1995,18(5):646～653. 被引量：1
10[7] Liang P, Hughes V, Fukagawa N K. Increased prevalence of mitochondrial DNA deletions in skeletal muscle of older individuals with impaired glucose tolerance: possible marker of glycemic stress[J]. Diabetes， 1997, 46(5):920～923. 被引量：1
