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心肌缺血再灌注损伤保护剂研究进展 被引量:4

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摘要 心肌缺血再灌注损伤(MIRI)的发病机制包括氧自由基损伤、钙超载、心肌能量代谢异常、内源性心脏保护物质作用、钾通道及其受体作用、以及细胞凋亡等因素参与。抗MIRI的药物通过上述机制而产生心肌保护作用。
出处 《实用预防医学》 CAS 2008年第2期616-618,共3页 Practical Preventive Medicine
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参考文献17

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二级参考文献58

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