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STAT5在哮喘发病机制中的作用 被引量:2

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作者 杜娟 邱晨
出处 《国外医学(呼吸系统分册)》 2005年第9期650-652,共3页 Section of Respiratory System Foreign Medical Sciences
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  • 2Buitenhuis M, Baltus B. Singal transducer and activator of transcription 5a(STAT5a) is required for eosinophil differention of human cord blood-derived CD34+ cells. Blood, 2003,101 ( 1 ):134-142. 被引量:1
  • 3Soldaini E,John S, Moro S, et al. DNA binding site selection of dimeric and tetrameric Stat5 proteins reveals a large repertoire of divergent tetrameric Stat5a binding sites. Mol Cell Biol, 2000,20(1) :389-401. 被引量:1
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同被引文献28

  • 1李昌崇,叶乐平,苏苗赏,李孟荣,张海邻,蔡晓红,董琳,罗运春.黄芪对哮喘大鼠肺组织信号转导子和转录激活子4表达的调控[J].中华儿科杂志,2007,45(10):727-731. 被引量:11
  • 2Darnell JE Jr. STATs and gene regulation. Science, 1977,277 (5332) : 1630-1635. 被引量:1
  • 3Barnes PJ, Adcock IM. Transcription factors and asthma. Eur Respir J, 1998,12( 1 ) :221-234. 被引量:1
  • 4Schindler CW. Sereis introduction:JA K-STAT signaling in hu- man disease. Clin Inverst ,2002,109 ( 9 ) : 1133-1137. 被引量:1
  • 5Guo FH, Comhair SA, Zheng S, et al. Molecular mechanisms of increased nitric in asthma:evidence for transcriptional and postt- ranslational regulation of NO synthesis. J Immunol, 2000, 164 ( 11 ) :5970-5980. 被引量:1
  • 6Quarcoo D,Weixler S, Groneberg D, et al. Inhibition of signal transducer and activator of transcription 1 attenuates allergen in- duced airway inflammation and hyperreactivity. J Allergy Clin Immunol, 2004,114 ( 2 ) :288-295. 被引量:1
  • 7Simeone-Penney MC, Severgnini M, Rozo L, et al. PDGF-in- duced human air way smooth muscle cell proliferation requires STAT3 and the small GTPase Racl. Am J Physiol Lung Cell Mol P hysiol,2008,294(4) :L698-704. 被引量:1
  • 8Simeone-Penney MC, Severgnini M, Tu P, et al. Airway epithe- lial STAT3 is required for allergic inflammation in a murine model of asthma. J Immunol,2007,178(10) :6191-6199. 被引量:1
  • 9Pinto LA, Steudemann L, Depner M, et al. STAT1 gene varia- tions,IgE regulation and atopy. Allergy, 2007,62 ( 12 ) : 1456- 1461. 被引量:1
  • 10Matsubara S,Takeda K,Kodama T,et al. IL-2 and IL-18 attenu- ation of airway hyperresponsiveness requires STAT4, IFN-gam- ma, and natural killer cells. Am J Respir Cell Mol Biol,2007,36 (3) :324-332. 被引量:1

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