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脊髓纵裂神经缺陷机制探讨

Study of Mechanism of Neural Deficit in Diastematomyelia
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摘要 对29例脊髓纵裂的神经功能状态进行临床评价及胫后神经皮层体感诱发电位(PTNCSEP)检查,结果:患病组29例两下肢间神经缺陷的临床评分及PTNCSEP有显著差异(P<0.05);患病组29例的58根PTNCSEP明显异常;手术组20例间隔切除术后神经缺陷的临床评分及PTNCSEP异常明显改善(P<0.05),非手术组9例1年半随访时则无改善(P>0.O5)。结果提示神经缺陷的机理是脊髓纵裂间隔对脊髓的牵拉和纵裂脊髓单侧发育异常。 29 patients with diastematomyelia ( 20 patients in operative group, 9 patients in conservative group). were studied by evaluating clinical neural function and measuring posterior tibial nerve cortical somatosensory evoked potentials (PTNCSEP). The results were that clinical scores and PTNCSEP had significant difference between bilateral lower extremities in 29 patients(P< 0. 05). Clinical scores and PTNCSEP improved significantly in operative group(P<0.05), and had not significant improvement in conservative group(P>0. 05). It showed that the mechanism of neural deficit was traction of the septum to the spinal cord and asymmetrical developmental anomalies of the cord.
出处 《中国矫形外科杂志》 CAS CSCD 1995年第4期242-243,共2页 Orthopedic Journal of China
关键词 脊髓纵裂 神经缺陷 外科手术 Diastematomyelia, Cortical somatosensory evoked potential , Operative treatment.
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