摘要
利用免疫组织化学方法对幽门螺杆菌(HP)感染胃粘膜内IgA,IgG和IgM产生细胞进行了研究.结果表明,HP感染引起了胃粘膜内局部抗体产生细胞的增加.其中以IgG和IgM产生细胞增加更着.提示:HP感染时局部体液免疫反应增强,进一步为HP是引起慢性活动性胃炎的病原提供了依据.
IgA, IgG and IgM-producing cells in gastric mucosa with helicobacter pylori(HP) were studied imrnunohistochemistry. The result showed that HP caused increase of local antibodies-producing in gastric mucosa, especially IgG and IgM-pro-ducing cells. It is suggested that local humoral immunoreaction is heightened by HP infection. This provides the evidence further that HP is one of the cauese of chronic active gastritis.
出处
《临床消化病杂志》
1994年第4期157-158,165,共3页
Chinese Journal of Clinical Gastroenterology
关键词
幽门螺杆菌
胃炎
免疫组织化学
Helicobacter pylori Chronic gastritis Immunohistochemistry
