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MMP-9与冠状动脉粥样硬化 被引量:8

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出处 《临床心血管病杂志》 CAS CSCD 北大核心 2004年第11期699-701,共3页 Journal of Clinical Cardiology
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  • 1[1]Galis Z S, Sukhova G K, Lark M W, et al. Increased expression of matrix metalloproteinases and matrix degrading activity in vulnerable regions of human atherosclerotic plaques. J Clin Invest, 1994, 94:2493-2503. 被引量:1
  • 2梁晓萍,戴勇,石丹,徐宜英.血清基质金属蛋白酶-9与冠心病相关性的临床研究[J].中国心血管杂志,2002,7(2):94-95. 被引量:24
  • 3[3]Galis Z S, Muszynski M, Sukhova G K, et al. Cytokine-stimulated human vascular smooth muscle cells synthesize a complement of enzymes required for extracellura matrix digestion. Circ Res , 1994, 75 : 181-189. 被引量:1
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  • 6[6]Mason D P, Kenagy R D, Hasenstab D, et al. Matrix metalloproteinase- 9 over expression enhance vascular smooth muscle cell migration and alters remodeling in the injured rat carotid artery. J Circ Res, 1999, 85:1179-1185. 被引量:1
  • 7[7]Denis G, Eugen I, Chad J, et al. Remodeling of Carotid Artery is associated with increased expression of matrix metalloprotenases in mouse blood flow cessation model. Circulation, 2000, 102: 2861-2867. 被引量:1
  • 8[8]Zorina S, Asanuma K, Godin D, et al. N-Acetyl-Cystine decreases the matrix-degrading capacity of macrophage-derived foam cells. New target for antioxidant therapy. Circulation, 1998, 97: 2445-2453. 被引量:1
  • 9[9]Brown D L, Hibbs M S, Kearney M, et al. Identification of 92-kD gelatinase in human coronary atherosclerotic lesions. Association of active enzyme synthesis with unstable angina. Circulation, 1995, 91: 2125-2131. 被引量:1
  • 10[10]Peterson M, Porter K E, Loftus I M, et al. Marimastat inhibits neointimal thickening in a model of human arterial intimal hyperplasia. Eur J Vase Endovase Surg, 2000, 19: 461-467. 被引量:1

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