1Robicsek F, Thubrikar MJ, Fokin AA. Cause of degenerative disease of the trileaflet aortic valve. Ann Thorac Surg, 2002, 73: 1346-1354. 被引量:1
2Moenckeberg JG. Der normale histologische Bau und die Sklerose der Aortenklappen. Virchows Arch Pathol Anat,1904,176:472-514. 被引量:1
3Roberts WC. Anatomically isolated aortic valvular disease. The case against its being of rheumatic etiology. Am J Med,1970,49:151-159. 被引量:1
4Soler-Soler J,Galve E. Worldwide perspective of heart valve disease. Heart,2000,83: 721-725. 被引量:1
5Pang DC,Choo SJ,Luo HH, et al. Significant increase of aortic root volume and commissural area occurs prior to aortic valve opening. J Heart Valve Dis,2000:9: 9-15. 被引量:1
6Stewart BF,Siscovick D,Lind BK,et al. Clinical factors associated with calcific aortic valve disease. Cardiovascular Health Study. J Am Coll Cardiol,1997:29:630-634. 被引量:1
7Otto CM, Kuusisto J, Reichenbach DD, et al. Characterization of the early lesion of ''degenerative'' valvar aortic stenosis. Histological and immunohischemical studies. Circulation,1995,92: 2163-2168. 被引量:1
8Melvin D. Cheitlin, MD. Pathophysiology of valvular aortic stenosis in the elderly. Am J geriatri cardiol, 2003, 12:173-177. 被引量:1
9O'Brien KD, Kuusisto J, Reichenach DD, et al. Osteopontin is expressed in human aortic valve lesions. Circulation,1995,92: 2163-2168. 被引量:1
10Roberts WC. The senile cardiac calcification syndrome. Am J Cardiol,1986,58:572-574. 被引量:1