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创伤性硬膜下积液转化为慢性硬膜下血肿危险因素分析 被引量:11

Analysis of risk factors for transformation of traumatic subdural effusion into chronic subdural hematoma
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摘要 目的分析创伤性硬膜下积液转化为慢性硬膜下血肿的危险因素。方法 2017年12月至2019年7月共治疗75例颅脑创伤伴创伤性硬膜下积液患者,通过CT或MRI观察治疗与随访期间硬膜下积液是否转化为慢性硬膜下血肿,分析相关影响因素,单因素和多因素逐步法Logistic回归分析筛查创伤性硬膜下积液转化为慢性硬膜下血肿的危险因素。结果 Logistic回归分析显示,硬膜下积液密度高于脑脊液密度(OR=6.021,95%CI:1.047~34.631;P=0.044)、创伤后达最大积液量时间长(OR=1.253,95%CI:1.002~1.568;P=0.048)、最大积液厚度大(OR=2.080,95%CI:1.267~3.414;P=0.004)和并发脑萎缩(OR=35.392,95%CI:1.397~896.474;P=0.031)是创伤性硬膜下积液转化为慢性硬膜下血肿的危险因素。结论创伤后积液长期处于进展期、积液密度高于脑脊液密度、最大积液厚度较大、并发脑萎缩的创伤性硬膜下积液患者易转化为慢性硬膜下血肿。 Objective To analyze the risk factors of traumatic subdural effusion(TSE) transforming into chronic subdural hematoma(CSDH).Methods A total of 75 patients with TSE after traumatic brain injury(TBI) were included from December 2017 to July 2019.CT or MRI reexamination during treatment and follow-up were performed to observe whether CSDH was transformed and relevant influencing factors were analyzed.Univariate and multivariate Logistic regression analyses were performed to screen the risk factors for the transformation of TSE into CSDH.Results Logistic regression analysis showed that the higher effusion density(OR=6.021,95% CI:1.047-34.631;P=0.044),subdural effusion increases for a long time(OR=1.253,95% CI:1.002-1.568;P=0.048),the thicker effusion(OR=2.080,95% CI:1.267-3.414;P=0.004) and brain atrophy(OR=35.392,95% CI:1.397-896.474;P=0.031) were risk factors for the transformation of TSE into CSDH.Conclusions The subdural effusion increases for a long time,higher effusion density,thicker effusion and brain atrophy are the risk factors associated with the transformation of TSE into CSDH.
作者 易盼 王存祖 陈品 王东东 王辉 何世维 曾巍 YI Pan;WANG Cun⁃zu;CHEN Pin;WANG Dong⁃dong;WANG Hui;HE Shi⁃wei;ZENG Wei(Graduate School,Dalian Medical University,Dalian 116044,Liaoning,China;Department of Neurosurgery,Northern Jiangsu People's Hospital,Yangzhou 225001,Jiangsu,China)
出处 《中国现代神经疾病杂志》 CAS 北大核心 2020年第7期602-607,共6页 Chinese Journal of Contemporary Neurology and Neurosurgery
关键词 脑损伤 创伤性 硬膜下积液 血肿 硬膜下 危险因素 LOGISTIC模型 Brain injuries,traumatic Subdural effusion Hematoma,subdural Risk factors Logistic models
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  • 1高乃康,毛伯镛.慢性硬膜下血肿治疗及其复发相关因素研究[J].中华神经外科疾病研究杂志,2004,3(4):350-351. 被引量:24
  • 2陶志强,朱志刚,陈相兵,胡茂通,丁胜鸿,楼洪福,方激杨,黄建跃.外伤性硬膜下积液演变为血肿机制研究[J].浙江创伤外科,2005,10(1):17-18. 被引量:31
  • 3陶志强,惠国桢,朱志刚,胡茂通,丁胜鸿.外伤性硬脑膜下积液转化成慢性硬脑膜下血肿的可能机制[J].江苏医药,2006,32(6):534-536. 被引量:21
  • 4陶志强,朱志刚,丁胜鸿,方激扬,陈相兵.急性硬膜下血肿保守治疗的一个前瞻性研究[J].临床医学,2007,27(2):17-19. 被引量:9
  • 5Mayfrank L, Laborde G, Lippitz B, et al. Bilateral chronic subdural hematoma following traumatic cerebrospinal fluid leakage into the thoracic epidural space [ J ]. Actda Neurochir (Wien) , 1993, 120 ( 1 - 2) : 92 -94. 被引量:1
  • 6Shono T, Inamura T, Morioka T, et al. Vascular endothelial growth factor in chronic subdural haematomas [ J]. J Clin Neurosci, 2001, 8 (5) : 411 -415. 被引量:1
  • 7Hohenstein A, Erber R, Schilling L, et al. Increased mRNA expression of VEGF within the hematoma and imbalance of angiopoietin-1 and -2 mRNA within the neomembranes of chronic subdural Hematoma[ J ]. J Neurotrauma, 2005, 22 ( 5 ) : 518 - 528. 被引量:1
  • 8Suzuki M, Kudo A, Kitakami A, et al. Local hypercoagulative activety precedes hyperfibrinolytic activity in the subdural space during development of chronic subdural haematoma from subdural effusion [J]. ActaNeurochir (Wien), 1998, 140(3): 261 -266. 被引量:1
  • 9Suzuki M, Endo S, Inada K, et al. Inflammatory cytokines locally elevated in chronic subdural haematoma [ J]. Aeta Neurochir (Wien) , 1998, 140(1) : 51 -55. 被引量:1
  • 10Lee KS, Bee WK, Doh JW, et al. Origin of chronic subdural haematoma and relation to traumatic subdural lesions [ J ]. Brain Inj, 1998, 12(11) : 901 -910. 被引量:1

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