分析了外源10μmol/L茉莉酸甲酯(M ethyl jasm on ate,MeJA)熏蒸处理对"巨峰"葡萄果穗在1℃贮藏期间离区膜脂过氧化及果实落粒的影响。结果表明,10μmol/LMeJA处理可显著降低葡萄果实冷藏期间落粒率、腐烂率和褐变指数,维持...分析了外源10μmol/L茉莉酸甲酯(M ethyl jasm on ate,MeJA)熏蒸处理对"巨峰"葡萄果穗在1℃贮藏期间离区膜脂过氧化及果实落粒的影响。结果表明,10μmol/LMeJA处理可显著降低葡萄果实冷藏期间落粒率、腐烂率和褐变指数,维持果实感官品质,同时显著诱导贮藏期间葡萄果穗离区组织中活性氧代谢酶(SOD、CAT、APX和POD)活性的上升并抑制超氧阴离子和羟基自由基生成量的增加。经MeJA处理的离区细胞膜中不饱和脂肪酸亚油酸和亚麻酸含量明显高于对照水平,而饱和脂肪酸棕榈酸和硬脂酸含量、相对电导率和MDA含量却显著低于对照水平。这些结果说明,MeJA处理可通过提高葡萄离区组织中活性氧代谢酶活性来减慢离区膜脂过氧化进程并维持离区细胞结构,从而降低了贮藏期间葡萄果实落粒率,改善了果实品质。展开更多
Jasmonic acid （JA） is a natural hormone regulator involved in development, responses against woundingand pathogen attack. Upon perception of pathogens, JA is synthesized and mediates a signaling cascade initiating v...Jasmonic acid （JA） is a natural hormone regulator involved in development, responses against woundingand pathogen attack. Upon perception of pathogens, JA is synthesized and mediates a signaling cascade initiating various defense responses. Traditionally, necrotrophic fungi have been shown to be the primary activators of JA- dependent defenses through the JA-receptor, COIl. Conversely, plants infected with biotrophic fungi have classically been associated with suppressing JA-mediated responses. However, recent evidence has shown that certain biotrophic fungal species also trigger activation of JA-mediated responses and mutants deficient in JA signaling show an increase in susceptibility to certain biotrophic fungal pathogens. These findings suggest a new role for JA in defense against fungal biotrophs. This review will focus on recent research advancing our knowledge of JA-dependant responses involved in defense against both biotrophic and necrotrophic fungi.展开更多
文摘分析了外源10μmol/L茉莉酸甲酯(M ethyl jasm on ate,MeJA)熏蒸处理对"巨峰"葡萄果穗在1℃贮藏期间离区膜脂过氧化及果实落粒的影响。结果表明,10μmol/LMeJA处理可显著降低葡萄果实冷藏期间落粒率、腐烂率和褐变指数,维持果实感官品质,同时显著诱导贮藏期间葡萄果穗离区组织中活性氧代谢酶(SOD、CAT、APX和POD)活性的上升并抑制超氧阴离子和羟基自由基生成量的增加。经MeJA处理的离区细胞膜中不饱和脂肪酸亚油酸和亚麻酸含量明显高于对照水平,而饱和脂肪酸棕榈酸和硬脂酸含量、相对电导率和MDA含量却显著低于对照水平。这些结果说明,MeJA处理可通过提高葡萄离区组织中活性氧代谢酶活性来减慢离区膜脂过氧化进程并维持离区细胞结构,从而降低了贮藏期间葡萄果实落粒率,改善了果实品质。
文摘Jasmonic acid （JA） is a natural hormone regulator involved in development, responses against woundingand pathogen attack. Upon perception of pathogens, JA is synthesized and mediates a signaling cascade initiating various defense responses. Traditionally, necrotrophic fungi have been shown to be the primary activators of JA- dependent defenses through the JA-receptor, COIl. Conversely, plants infected with biotrophic fungi have classically been associated with suppressing JA-mediated responses. However, recent evidence has shown that certain biotrophic fungal species also trigger activation of JA-mediated responses and mutants deficient in JA signaling show an increase in susceptibility to certain biotrophic fungal pathogens. These findings suggest a new role for JA in defense against fungal biotrophs. This review will focus on recent research advancing our knowledge of JA-dependant responses involved in defense against both biotrophic and necrotrophic fungi.
