摘要
水分胁迫及ＡＢＡ处理可诱导丰抗８号小麦幼苗及其悬浮培养细胞中４４．２ＫＤ蛋白亚基的产生或大量合成。高浓度的Ｃａ２＋通道阻塞剂（Ｖｅｒａｐａｍｉｌ）处理丰抗８号小麦幼苗，可明显抑制水分胁迫诱导产生的４４．２ＫＤ蛋白亚基的大量合成，而悬浮培养细胞中，２０μＭ的异博定处理就可显著抑制由ＡＢＡ及ＡＢＡ＋ＰＥＧ所诱导的蛋白亚基含量的提高，说明Ｃａ２＋可能参与了干旱信号在胞内的传递过程；细胞对Ｃａ２＋通道阻塞剂的反应比幼苗更显著、更敏感。
Production or mass synthesis of 44.2 KD subunit in wheat seedlings and suspended culture cell of Feng Kang 8 was induced by water stress or ABA. Synthetic of 44.2 KD subunit in seedling of Feng Kang 8 was remarkably inhibited by highly concentrated Verapamil of Ca 2+ . The increase of 44.2 KD subunit content induced by ABA and ABA+PEG in suspension culture was obviously restrained by 20 μm verapamil. It demonstrated that Ca 2+ might involved in drought singal transmitting in cell, moreover the reaction of cell to ca 2+ passageway inhibitor was more remarkable and more sensitive than that of seedling.
出处
《干旱地区农业研究》
CSCD
北大核心
1999年第1期72-77,共6页
Agricultural Research in the Arid Areas
基金
农业部"八五"应用基础研究项目
关键词
冬小麦
水分胁迫
干旱诱导蛋白
CA^2+
wheat
water stress
drought induced protein
Ca 2+
Ca 2+ passageway inhibitor
